Dexmedetomidine Might Exacerbate Acute Kidney Injury, While Midazolam Might Have a Postconditioning Effect: A Rat Model of Lipopolysaccharide-Induced Acute Kidney Injury
Author:
Affiliation:
1. Department of Anesthesiology and Pain Medicine, Graduate School of Medicine, Nippon Medical School
Publisher
Medical Association of Nippon Medical School
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/jnms/90/5/90_JNMS.2023_90-406/_pdf
Reference33 articles.
1. 1. Fleischmann C, Scherag A, Adhikari NK, et al. Assessment of global incidence and mortality of hospital-treated sepsis. Current estimates and limitations. Am J Respir Crit Care Med. 2016 Feb 1;193 (3):259-72.
2. 2. Gomez H, Kellum JA. Sepsis-induced acute kidney injury. Curr Opin Crit Care. 2016 Dec;22 (6):546-53.
3. 3. Yasuda H, Kato A, Fujigaki Y, Hishida A; Shizuoka Kidney Disease Study Group. Incidence and clinical outcomes of acute kidney injury requiring renal replacement therapy in Japan. Ther Apher Dial. 2010 Dec;14 (6):541-6.
4. 4. Matsuda A, Jacob A, Wu R, et al. Novel therapeutic targets for sepsis: regulation of exaggerated inflammatory responses. J Nippon Med Sch. 2012;79 (1):4-18.
5. 5. Chen Y, Luan L, Wang C, et al. Dexmedetomidine protects against lipopolysaccharide-induced early acute kidney injury by inhibiting the iNOS/NO signaling pathway in rats. Nitric Oxide. 2019 Apr 1;85:1-9.
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