Subclinical Association of Aortic Stiffness with Cardiac Structure and Function in African- Americans: the Jackson Heart Study

Author:

Jha Mawra1,Musani Solomon2,Mccarthy Inbar1,Hundley W. Gregory3,Carr J. Jeffrey4,Terry James G.3,Oshunbade Adebamike2,Vasan Ramachandran S.5,Butler Javed2,Hall Michael2,Mitchell Gary F.6,Fox Ervin2,Tsao Connie W.1

Affiliation:

1. Beth Israel Deaconess Medical Center, Harvard Medical School

2. University of Mississippi Medical Center

3. Virginia Commonwealth University Medical Center

4. Vanderbilt University Medical Center

5. Boston University School of Medicine, Boston University School of Public Health

6. Cardiovascular Engineering Inc

Abstract

Abstract

Background Cardiovascular disease (CVD) morbidity and mortality are high among black adults. We aimed to study the granular subclinical relations of aortic stiffness and left ventricular (LV) function and remodeling in blacks, in whom limited data are available. Methods In the Jackson Heart Study, 1,050 U.S. community-dwelling black adults without CVD (mean age 59±10 years, 62% women) underwent 1.5T cardiovascular magnetic resonance. We assessed regional and global aortic stiffness and LV structure and function, including LV mass indexed to body surface area (LVMI), end-diastolic volume (LVEDV), ejection fraction (EF), and global and regional circumferential strain (Ecc). Results Phase contrast images of the cross-sectional aorta at the pulmonary artery bifurcation and abdominal aorta bifurcation were acquired to measure pulse wave velocity of the aortic arch (AA-PWV) and thoracic aorta (T-PWV). Results of multivariable-adjusted analyses are presented as SD unit change in LV variables per SD change in PWV variables. Higher AA-PWV and T-PWV were associated with greater LVMI: for T-PWV, β=0.10, 95% CI=0.03-0.16, p=0.002. Higher AA-PWV and T-PWV were associated with worse (more positive) Ecc at the LV base (for AA-PWV, β=0.13, 95% CI=0.05-0.20, p=0.0007), but not mid-LV or apex. AA-PWV and T-PWV were not associated with LV mass/LVEDV or EF. Conclusions In this cross-sectional study of blacks without CVD in the U.S., aortic stiffness is associated with subclinical adverse LV function in basal segments. Future studies may elucidate the temporal relationships of aortic stiffness on the pattern and progression of LV remodeling, dysfunction, and associated prognosis in blacks.

Publisher

Research Square Platform LLC

Reference35 articles.

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2. Kass SJZ, Vojtech M, Mechanisms DA (2005) Pathophysiology, and Therapy of Arterial Stiffness. 10.1161/01.ATV.0000160548.78317.29

3. Wentland AL, Grist TM, Wieben O Review of MRI-based measurements of pulse wave velocity: a biomarker of arterial stiffness. Cardiovasc Diagn Ther. 42014. pp. 193–206

4. Prognostic value of aortic pulse wave velocity as index of arterial stiffness in the general population;Willum-Hansen T;Circulation,2006

5. 1161/CIRCULATIONAHA 105.579342. PubMed PMID: 16461839

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