Extracellular vesicle-mediated Transfer of miRNA-1 from Primary tumor Repress Distant Metastasis Growth

Author:

Cho Je-Yoel1ORCID,Kim Hui-Su1,Lee Kang-Hoon1,Son Keun-Hong1,Shin Tae-Jin1

Affiliation:

1. Seoul National University

Abstract

Abstract Metastases originate from primary tumors and conquer distant organs. Growing evidence suggest that metastases are under control of primary tumors even out of the primary territory, however, how primary tumors remotely control metastases remains unclear. Here, we discovered a molecular mechanism by which a primary tumor suppresses the metastatic growth. Interestingly, we found that extracellular vesicles (EVs) derived from a primary tumor has an ability to inhibit the growth of metastases in vitro and in vivo. miR-1 was particularly enriched in the primary tumor-derived EVs (pTDEs) and responsible for the suppression of metastatic growth. Mechanistically, intracellular ROS and DNA damage were induced, which led to the cell cycle arrest. Collectively, our data demonstrate that the primary tumor restricts distant metastatic cancer by miR-1 in pTDEs which can potentially be used as an anticancer drug for metastasis.

Publisher

Research Square Platform LLC

Reference50 articles.

1. Primary tumor associated macrophages activate programs of invasion and dormancy in disseminating tumor cells;Borriello L;Nature communications,2022

2. Primary tumors release ITGBL1-rich extracellular vesicles to promote distal metastatic tumor growth through fibroblast-niche formation;Ji Q;Nature communications,2020

3. Primary tumor-induced immunity eradicates disseminated tumor cells in syngeneic mouse model;Piranlioglu R;Nature communications,2019

4. I. The dormant cancer cell life cycle;Phan TG;Nature Reviews Cancer,2020

5. Tumor dormancy in vivo by prevention of neovascularization;Gimbrone MA;The Journal of experimental medicine,1972

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