Angiotensin 1-7 reduces lipid deposition in the renal tubules of high-fat fed mice

Author:

qin Zheng1,Cui Hongqing1,Zhu Huanhuan1,Guo Honglei1

Affiliation:

1. The First Affiliated Hospital of Nanjing Medical University (Jiangsu Province Hospital)

Abstract

Abstract

Background To investigate the effects of angiotensin 1–7 (Ang-(1–7)) on proximal tubules in mice fed a high-fat diet (HFD). Methods Mice were randomly divided into three groups, including the control group (mice fed a standard rodent chow diet), HFD group, and HFD group treated with Ang-(1–7). At the end of the experiment, 24-h urine samples and kidney specimens were collected. We evaluated proximal tubule injury with PAS. Renal Oil Red O staining and immunofluorescence staining were used to disclose the expression of lipid deposition. Endoplasmic reticulum stress, inflammation and apoptosis were tested by Western blotting. Results Serum creatinine, blood urea nitrogen, and urinary albumin were elevated in HFD mice, while urinary albumin was decreased after Ang-(1–7) treatment. Ang-(1–7) dramatically inhibited the development of vacuolated tubular cells and lipid deposition while decreasing the expression of perilipin-2 and CD36. Ang-(1–7) also ameliorated the increase in endoplasmic reticulum stress and apoptosis. Furthermore, increased TNF-α, MCP-1, and IL-1β levels in HFD mice were inhibited by Ang-(1–7) treatment. Conclusions Ang-(1–7) treatment mediated reno-protection by attenuating lipotoxicity to inhibit inflammation and endoplasmic reticulum stress-induced apoptosis in HFD mice. These findings may offer a novel therapy for HFD-related renal injury.

Publisher

Research Square Platform LLC

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