Carvacrol and Thymol Attenuate Cytotoxicity Induced by Amyloid β25-35 Via Activating Protein Kinase C and Inhibiting Oxidative Stress in PC12 Cells-Reference-Cited by-同舟云学术

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Carvacrol and Thymol Attenuate Cytotoxicity Induced by Amyloid β25-35 Via Activating Protein Kinase C and Inhibiting Oxidative Stress in PC12 Cells

Published:2020-07-01 Issue:4 Volume:24 Page:243-250
ISSN:1028-852X
Container-title:Iranian Biomedical Journal
language:en
Short-container-title:ibj

Author:

Azizi Zahra,Salimi Mona,Amanzadeh Amir,Majelssi Nahid,Naghdi Nasser, , , , ,

Publisher

CMV Verlag

Subject

Biochemistry, medical,Clinical Biochemistry,General Biochemistry, Genetics and Molecular Biology

Link

http://ibj.pasteur.ac.ir/article-1-3022-en.pdf

Reference46 articles.

1. Alzheimer's disease results from the cerebral accumulation and cytotoxicity of amyloid ß-protein

2. Iridoids and Other Monoterpenes in the Alzheimer’s Brain: Recent Development and Future Prospects

3. Classification and basic pathology of Alzheimer disease

4. Targeting of cell-surface β-amyloid precursor protein to lysosomes: alternative processing into amyloid-bearing fragments

5. The cell biology of β-amyloid precursor protein and presenilin in Alzheimer's disease

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2. Natural polyphenol: Their pathogenesis-targeting therapeutic potential in Alzheimer's disease;European Journal of Medicinal Chemistry;2024-04

3. Thymol as a Potential Neuroprotective Agent: Mechanisms, Efficacy, and Future Prospects;Journal of Agricultural and Food Chemistry;2024-03-20

4. The Neuroprotective Properties of Thymol and Carvacrol: A Review Study;The Natural Products Journal;2024-02-29

5. Effect of thymol on oxidative stress and reelin signaling pathway in Alzheimer's disease model;Turkish Journal of Biology;2024-02-27

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