Voluntary Physical, Not Forced Exercise, Ameliorates Tau Pathology and Cognition in Tauopathy Model Mice
Author:
Affiliation:
1. Department of Diagnosis, Prevention and Treatment of Dementia, Juntendo University Graduate School of Medicine
2. Department of Neurology, Juntendo University Graduate School of Medicine
Publisher
The Jutendo Medical Journal
Link
https://www.jstage.jst.go.jp/article/jmj/64/Suppl.1/64_JMJ18-P36/_pdf
Reference16 articles.
1. 1) Prince M, Comas-Herrera A, Knapp M, Guerchet M, Karagiannidou M: World Alzheimer Report 2016, Improving healthcare for people living with dementia. (https://www.alz.co.uk/research/WorldAlzheimerReport2016.pdf)
2. 2) Grundke-Iqbal I, Iqbal K, Tung YC, Quinlan M, Wisniewski HM, Binder LI: Abnormal phosphorylation of the microtubule-associated protein tau (tau) in Alzheimer cytoskeletal pathology. Proc Natl Acad Sci U S A, 1986; 83: 4913-4917.
3. 3) Larson EB, Wang L, Bowen JD, et al: Exercise is associated with reduced risk for incident dementia among persons 65 years of age and older. Ann Intern Med, 2006; 144: 73-81.
4. 4) Pasinetti GM, Eberstein JA: Metabolic syndrome and the role of dietary lifestyles in Alzheimer’s disease. J Neurochem, 2008; 106: 1503-1514.
5. 5) Laurin D, Verreault R, Lindsay J, MacPherson K, Rockwood K: Physical activity and risk of cognitive impairment and dementia in elderly persons. Arch Neurol, 2001; 58: 498-504.
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