Prenatal Exposure to Di(2-ethylhexyl) phthalate and Subsequent Infant and Child Health Effects-Reference-Cited by-同舟云学术

Prenatal Exposure to Di(2-ethylhexyl) phthalate and Subsequent Infant and Child Health Effects

Published:2015 Issue:3 Volume:3 Page:70-83
ISSN:2187-8404
Container-title:Food Safety
language:en
Short-container-title:Food Safety

Author:

Nakajima Tamie¹,Hayashi Yumi²³,Ito Yuki⁴

Affiliation:

1. College of Life and Health Sciences, Chubu University, 1200 Matsumoto-cho, Kasugai 487-8501, Japan

2. Department of Radiological and Medical Laboratory Sciences, Nagoya University Graduate School of Medicine, 1-1-20 Daiko-Minami, Higashi-ku, Nagoya 461-8673, Japan

3. Institute for Advanced Research, Nagoya University, Furo-cho, Chikusa-ku, Nagoya 464-8601, Japan

4. Department of Occupational and Environmental Health, Nagoya City University Graduate School of Medical Sciences, 1 Kawasumi, Mizuho-cho, Mizuho-ku, Nagoya 467-8601, Japan

Publisher

Food Safety Commission, Cabinet Office

Link

https://www.jstage.jst.go.jp/article/foodsafetyfscj/3/3/3_2015011/_pdf

Reference75 articles.

1. 1. Japan Plasticizer Industry Association. Available at: http://www.kasozai.gr.jp/data/toukei-pdf/2013-07seisan.pdf.

2. 2. International Agency for Research on Cancer. Di(2-ethylhexyl)phthalate. Some Chemicals Present in Industrial and Consumer Products, Food and Drinking-water. vol. 101. IARC.2012: 144–274. Monographs on the Evaluation of Carcinogenic Risks to Humans.

3. 3. Food Safety Commission of Japan. Risk Assessment Report Bis(2-ethylhexyl)phthalate (DEHP) (Apparatuses, Containers and Packages). Available at: http://www.fsc.go.jp/english/evaluationreports/apparatus_containers/risk_assessment_report.pdf.

4. 4. International Agency for Research on Cancer. Some Industrial Chemicals. IARC Monographs on the Evaluation of Carcinogenic Risks to Humans. Vol. 77. 1-529IARC. 2000. Available at: http://monographs.iarc.fr/ENG/Monographs/vol77/mono77-6.pdf.

5. 5. Ito Y, Yamanoshita O, Asaeda N, et al. Di(2-ethylhexyl)phthalate induces hepatic tumorigenesis through a peroxisome proliferator-activated receptor alpha-independent pathway. J Occup Health. 2007; 49(3): 172–182.

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