Interleukin‐1 and Tumor Necrosis Factor‐α Induce Collagenolysis and Bone Resorption by Regulation of Matrix Metalloproteinase‐2 in Mouse Calvarial Bone Cells-Reference-Cited by-同舟云学术

Interleukin‐1 and Tumor Necrosis Factor‐α Induce Collagenolysis and Bone Resorption by Regulation of Matrix Metalloproteinase‐2 in Mouse Calvarial Bone Cells

Published:2003-01 Issue:3 Volume:25 Page:347-364
ISSN:0892-3973
Container-title:Immunopharmacology and Immunotoxicology
language:en
Short-container-title:Immunopharmacology and Immunotoxicology

Author:

Kang Bong‐Seok,Park Young‐Guk,Cho Jin‐Young,Kim June‐Ki,Lee Tae‐Kyun,Kim Dong‐Wook,Gu Yeun‐Hwa,Suzuki Ikukatsu,Chang Young‐Chae,Kim Cheorl‐Ho

Publisher

Informa UK Limited

Subject

Pharmacology,Toxicology,General Medicine,Immunology,Immunology and Allergy

Link

http://www.tandfonline.com/doi/pdf/10.1081/IPH-120024503

Reference37 articles.

1. Tumor necrosis factor type α (cachectin) stimulates mouse osteoblast-like cells (MC3T3-E1) to produce macrophage-colony stimulating activity and prostaglandin E2

2. Stimulation of prostaglandin e2 production by interleukin-1α and transforming growth factor α in osteoblastic MC3T3-E1 cells

3. Mechanism of Regulation of Prostaglandin Production by Parathyroid Hormone, Interleukin-1, and Cortisol in Cultured Mouse Parietal Bones*

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