Author:
Gokhale Avanti,Freeman Amanda A. H.,Hartwig Cortnie,Bassell Julia L.,Zlatic Stephanie A.,Sapp Christie,Vadlamudi Trishna,Abudulai Farida,Crocker Amanda,Werner Erica,Wen Zhexing,Repetto Gabriela M.,Gogos Joseph A.,Claypool Steven M.,Forsyth Jennifer K.,Bearden Carrie,Gausier Jill,Lewis David A.,Seyfried Nicholas T.,Faundez Victor
Reference84 articles.
1. Each column is one zeitgeber hour and each row is one animal. C-H) Probability plots of sleep parameters per 24 hours (C, D and G) or 12 hours light/dark periods (E, F and H) from animals depicted in B. TST is total sleeping time. G-H) The number of sleep bouts is decreased in catecholaminergic-specific sea RNAi animals. No effect of glutamatergic-specific sea RNAi (VGlut>CS=38, sea RNAi=40, VGlut>RNAi= 44 animals) C-J) p values were estimated with the Kolmogorov-Smirnov test Supplementary Fig. 5. Comparative Bioinformatics of the 22q11.2 Proteome and Two Independent Df(16)A-/+ Brain Proteomes. A) Venn diagrams depict from top to bottom: a comparison of common hits between our Df(16)A-/+ brain proteome and the Df(16)A-/+ brain proteome reported by Wesseling et al. PMID: 27001617. The Wesseling Df(16)A-/+ brain proteome and our 22q11.2 proteome. The Wesseling Df(16)A-/+ brain proteome and the mouse Mitocarta 2.0 dataset. B) Cellular Component gene ontology analysis of GO CC generated with the ENRICHR engine using the Wesseling Df(16)A-/+ brain proteome dataset and a similarly sized random mouse gene dataset. Random gene list was generated with the engine RandomGeneSetGenerator. C) Cellular Component gene ontology analysis (GO CC) was performed with the ENRICHR engine using the Wesseling Df(16)A-/+ brain proteome dataset either by itself, or in combination with our 22q11.2 proteome, or with 1500 (1x) or 3000 (2x) randomly generated genes. See discussion;sea RNAi control (n=40), and catecholaminergic-specific sea RNAi animals (Ddc>RNAi, n=40) depict activity for each animal averaged across one hour bins
2. Related to Figure 2E-F. Quantitative Mass Spectrometry Data for Df(16)A-/+ brains. Supplementary Table 4. Related to Figure S1. Comparative Bioinformatic Analysis of 22q11.2 Genealogical Proteomes. Supplemental Table 5. Primers Used in these;S Pyl;Genealogical Proteomes and Df(16)A-/+ brain proteomes. Supplementary Table 3,2012
3. Mitochondrial Involvement in Psychiatric Illness in Adults
4. The psychiatric manifestations of mitochondrial disorders: a case and review of the literature;J Clin Psychiatry
5. The psychiatric presentation of mitochondrial disorders in adults;R E Anglin;J Neuropsychiatry Clin Neurosci,2012
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