Increased Expression of KIF11/Kinesin5 Offsets Alzheimer Aβ-Mediated Toxicity and Cognitive Dysfunction in Cell Culture, Mice, and Humans-Reference-Cited by-同舟云学术

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Increased Expression of KIF11/Kinesin5 Offsets Alzheimer Aβ-Mediated Toxicity and Cognitive Dysfunction in Cell Culture, Mice, and Humans

Published:2022 Issue: Volume: Page:
ISSN:1556-5068
Container-title:SSRN Electronic Journal
language:en
Short-container-title:SSRN Journal

Author:

Lucero Esteban,Freund Ronald,Smith Alexandra,Johnson Noah,Dooling Breanna,Sullivan Emily,Prikhodko Olga,Ahmed Md. Mahiuddin,Bennett David A.,Hohman Timothy,Dell'Acqua Mark,Chial Heidi,Potter Huntington

Publisher

Elsevier BV

Subject

General Earth and Planetary Sciences,General Environmental Science

Reference79 articles.

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2. Progressive, age-related behavioral impairments in transgenic mice carrying both mutant amyloid precursor protein and presenilin-1 transgenes;G W Arendash;Brain Res,2001

3. Alzheimer amyloid beta inhibition of Eg5/kinesin 5 reduces neurotrophin and/or transmitter receptor function;C Ari;Neurobiol Aging,2014

4. Kinesin molecular motor Eg5 functions during polypeptide synthesis;K M Bartoli;Mol Biol Cell,2011

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