Melatonin Improves Nitric Oxide Bioavailability in Isoproterenol Induced Myocardial Injury

Author:

Santos Ramison,Turck Patrick,de Mello Palma Victor,Visioli Fernanda,Ortiz Vanessa Duarte,Proença Isabel Cristina Teixeira,Fernandes Tânia Regina G.,Fernandes Elissa,Tasca Silvio,Carraro Cristina Campos,Belló-Klein Adriane,da Rosa Araujo Alex Sander,Khaper Neelam,de Castro Alexandre Luz

Publisher

Elsevier BV

Reference44 articles.

1. Simvastatin prevents isoproterenol-induced cardiac hypertrophy through modulation of the JAK/STAT pathway;N M Al-Rasheed;Drug Des Devel Ther,2015

2. Umbelliferone prevents isoproterenol-induced myocardial injury by upregulating Nrf2/HO-1 signaling, and attenuating oxidative stress, inflammation, and cell death in rats;O Y Althunibat;Biomed Pharmacother,2022

3. Translating from Proof-of-Concept Studies to Therapeutic Use;O C Baltatu;Int J Mol Sci,2019

4. Melatonin influences NO/NOS pathway and reduces oxidative and nitrosative stress in a model of hypoxic-ischemic brain damage;S Blanco;Nitric Oxide,2016

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