ATR signaling at a glance
Author:
Affiliation:
1. Massachusetts General Hospital Cancer Center, Harvard Medical School, Charlestown, MA 02129, USA
2. Department of Pathology, Harvard Medical School, Boston, MA 02115, USA
Publisher
The Company of Biologists
Subject
Cell Biology
Link
http://journals.biologists.com/jcs/article-pdf/122/3/301/1494907/301.pdf
Reference67 articles.
1. Ball, H. L., Myers, J. S. and Cortez, D. (2005). ATRIP binding to replication protein A-single-stranded DNA promotes ATR-ATRIP localization but is dispensable for Chk1 phosphorylation. Mol. Biol. Cell16, 2372-2381.
2. Ball, H. L., Ehrhardt, M. R., Mordes, D. A., Glick, G. G., Chazin, W. J. and Cortez, D. (2007). Function of a conserved checkpoint recruitment domain in ATRIP proteins. Mol. Cell. Biol.27, 3367-3377.
3. Bartek, J. and Lukas, J. (2003). Chk1 and Chk2 kinases in checkpoint control and cancer. Cancer Cell3, 421-429.
4. Bassermann, F., Frescas, D., Guardavaccaro, D., Busino, L., Peschiaroli, A. and Pagano, M. (2008). The Cdc14B-Cdh1-Plk1 axis controls the G2 DNA-damage-response checkpoint. Cell134, 256-267.
5. Berkovich, E., Monnat, R. J., Jr and Kastan, M. B. (2007). Roles of ATM and NBS1 in chromatin structure modulation and DNA double-strand break repair. Nat. Cell Biol.9, 683-690.
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