Outer mitochondrial membrane permeabilization during apoptosis triggers caspase-independent mitochondrial and caspase-dependent plasma membrane potential depolarization: a single-cell analysis
Author:
Affiliation:
1. Interdisciplinary Center for Clinical Research (IZKF), Westphalian Wilhelms-University, D-48149 Münster, Germany
2. Department of Pharmacology and Toxicology, Westphalian Wilhelms-University,D-48149 Münster, Germany
Abstract
Publisher
The Company of Biologists
Subject
Cell Biology
Link
http://journals.biologists.com/jcs/article-pdf/116/3/525/1363037/525.pdf
Reference55 articles.
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3. Bortner, C. and Cidlowski, J. (1999). Caspase independent/dependent regulation of K(+), cell shrinkage, and mitochondrial membrane potential during lymphocyte apoptosis. J. Biol. Chem.274,21953-21962.
4. Bortner, C. D., Gómez-Angelats, M. and Cidlowski, J. A. (2001). Plasma membrane depolarization without repolarization is an early molecular event in anti-Fas-induced apoptosis. J. Biol. Chem.276,4304-4314.
5. Bossy-Wetzel, E., Newmeyer, D. D. and Green, D. R.(1998). Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization. EMBO J.17, 37-49.
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