Bridging the myoplasmic gap II: more recent advances in skeletal muscle excitation–contraction coupling
Author:
Affiliation:
1. Department of Medicine-Cardiology Division, University of Colorado Denver-Anschutz Medical Campus, 12700 East 19th Avenue, Room 8006, B-139, Aurora, CO 80045, USA
Abstract
Publisher
The Company of Biologists
Subject
Insect Science,Molecular Biology,Animal Science and Zoology,Aquatic Science,Physiology,Ecology, Evolution, Behavior and Systematics
Link
http://journals.biologists.com/jeb/article-pdf/219/2/175/1309170/jeb124123.pdf
Reference93 articles.
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2. Intramembrane charge movements and excitation- contraction coupling expressed by two-domain fragments of the Ca2+ channel;Ahern;Proc. Natl. Acad. Sci. USA,2001
3. A component of excitation-contraction coupling triggered in the absence of the T671-L690 and L720-Q765 regions of the II-III loop of the dihydropyridine receptor α1S pore subunit;Ahern;Biophys. J.,2001
4. Twitches in the presence of ethylene glycol bis(β-aminoethyl ether)-N,N′-tetraacetic acid;Armstrong;Biochim. Biophys. Acta,1972
5. Bridging the myoplasmic gap: recent developments in skeletal muscle excitation–contraction coupling;Bannister;J. Muscle Res. Cell Motil.,2007
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