Alternative splicing affecting a novel domain in the C. elegansEGL-15 FGF receptor confers functional specificity
Author:
Affiliation:
1. Department of Cell Biology, Yale University School of Medicine, I-354 SHM PO Box 208005, New Haven, CT 06520-8005, USA
2. Department of Genetics, Yale University School of Medicine, I-354 SHM PO Box 208005, New Haven, CT 06520-8005, USA
Abstract
Publisher
The Company of Biologists
Subject
Developmental Biology,Molecular Biology
Link
http://journals.biologists.com/dev/article-pdf/130/16/3757/1496590/3757.pdf
Reference36 articles.
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2. Borland, C. Z., Schutzman, J. L. and Stern, M. J.(2001). Fibroblast growth factor signaling in Caenorhabditis elegans. BioEssays23,1120-1130.
3. Branda, C. S. and Stern, M. J. (2000). Mechanisms controlling sex myoblast migration in Caenorhabditis elegans hermaphrodites. Dev. Biol.226,137-151.
4. Brenner, S. (1974). The genetics of Caenorhabditis elegans. Genetics77, 71-94.
5. Burdine, R. D., Chen, E. B., Kwok, S. F. and Stern, M. J.(1997). egl-17 encodes an invertebrate fibroblast growth factor family member required specifically for sex myoblast migration in Caenorhabditis elegans. Proc. Natl. Acad. Sci. USA94,2433-2437.
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