Pre-mRNA splicing: life at the centre of the central dogma
Author:
Affiliation:
1. MRC Human Genetics Unit, Edinburgh, EH4 2XU, UK
Publisher
The Company of Biologists
Subject
Cell Biology
Link
http://journals.biologists.com/jcs/article-pdf/117/26/6261/1529840/6261.pdf
Reference37 articles.
1. Beyer, A. L. and Osheim, Y. N. (1988). Splice site selection, rate of splicing, and alternative splicing on nascent transcripts. Genes Dev.2, 754-765.
2. Blencowe, B. J. (2000). Exonic splicing enhancers: mechanism of action, diversity and role in human genetic diseases. Trends Biochem. Sci.25, 106-110.
3. Caceres, J. F. and Kornblihtt, A. R. (2002). Alternative splicing: multiple control mechanisms and involvement in human disease. Trends Genet.18, 186-193.
4. Cartegni, L. and Krainer, A. R. (2002). Disruption of an SF2/ASF-dependent exonic splicing enhancer in SMN2 causes spinal muscular atrophy in the absence of SMN1. Nat. Genet.30, 377-384.
5. de la Mata, M., Alonso, C. R., Kadener, S., Fededa, J. P., Blaustein, M., Pelisch, F., Cramer, P., Bentley, D. and Kornblihtt, A. R. (2003). A slow RNA polymerase II affects alternative splicing in vivo. Mol. Cell,12, 525-532.
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