Impairment of autophagy may represent the molecular mechanism behind the relationship between obesity and inflammation in patients with BPH and LUTS
Author:
Publisher
Edizioni Minerva Medica
Subject
General Medicine
Link
https://www.minervamedica.it/pdf.php?cod=R19Y2021N05A0631
Reference29 articles.
1. The influence of the medical treatment of LUTS on benign prostatic hyperplasia surgery: do we operate too late?
2. Autophagy deactivation is associated with severe prostatic inflammation in patients with lower urinary tract symptoms and benign prostatic hyperplasia
3. Obesity-associated inflammation induces androgenic to estrogenic switch in the prostate gland
4. Patients With Prostatic Inflammation Undergoing Transurethral Prostatic Resection Have a Larger Early Improvement of Storage Symptoms
5. Impaired macrophage autophagy increases the immune response in obese mice by promoting proinflammatory macrophage polarization
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3. Diagnostic efficacy of systemic immune-inflammation biomarkers in benign prostatic hyperplasia using receiver operating characteristic and artificial neural network;Scientific Reports;2023-09-08
4. MIR663AHG as a competitive endogenous RNA regulating TGF‐β‐induced epithelial proliferation and epithelial–mesenchymal transition in benign prostate hyperplasia;Journal of Biochemical and Molecular Toxicology;2023-07-30
5. Comparative RNA-sequencing analysis of the prostate in a mouse model of benign prostatic hyperplasia with bladder outlet obstruction;Molecular and Cellular Biochemistry;2023-03-15
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