Polo-Like Kinases (Plks), a Key Regulator of Cell Cycle and New Potential Target for Cancer Therapy
Author:
Funder
National Research Foundation of Korea
Publisher
Korean Society of Developmental Biology
Subject
General Medicine
Link
http://www.ksdb.org/download/download_pdf?pid=dr-18-1-65
Reference44 articles.
1. Polo-like kinase 1 (Plk1) inhibits p53 function by physical interaction and phosphorylation;Ando;J Biol Chem,2004
2. Managing the centrosome numbers game: from chaos to stability in cancer cell division;Brinkley;Trends Cell Biol,2001
3. Silencing of the novel p53 target gene Snk/Plk2 leads to mitotic catastrophe in paclitaxel (taxol)-exposed cells;Burns;Mol Cell Biol,2003
4. Polo-like kinase 1 regulates Nlp, a centrosome protein involved in microtubule nucleation;Casenghi;Dev Cell,2003
5. Plk2 regulates centriole duplication through phosphorylation-mediated degradation of Fbxw7 (human Cdc4);Cizmecioglu;J Cell Sci,2012
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