Role of B7H3/IL-33 Signaling in Pulmonary Fibrosis–induced Profibrogenic Alterations in Bone Marrow
Author:
Affiliation:
1. Department of Molecular and Internal Medicine, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima, Japan; and
2. Department of Pathology, University of Michigan Medical School, Ann Arbor, Michigan
Publisher
American Thoracic Society
Subject
Critical Care and Intensive Care Medicine,Pulmonary and Respiratory Medicine
Link
https://www.atsjournals.org/doi/pdf/10.1164/rccm.201808-1560OC
Reference48 articles.
1. Lung Bone Marrow–derived Hematopoietic Progenitor Cells Enhance Pulmonary Fibrosis
2. The In Vivo Fibrotic Role of FIZZ1 in Pulmonary Fibrosis
3. Bone Marrow CD11c+ Cell–Derived Amphiregulin Promotes Pulmonary Fibrosis
4. Identification of an atypical monocyte and committed progenitor involved in fibrosis
5. Donor Myocardial Infarction Impairs the Therapeutic Potential of Bone Marrow Cells by an Interleukin-1–Mediated Inflammatory Response
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