Protein Tyrosine Phosphatase-N13 Promotes Myofibroblast Resistance to Apoptosis in Idiopathic Pulmonary Fibrosis-Reference-Cited by-同舟云学术

Protein Tyrosine Phosphatase-N13 Promotes Myofibroblast Resistance to Apoptosis in Idiopathic Pulmonary Fibrosis

Published:2018-10 Issue:7 Volume:198 Page:914-927
ISSN:1073-449X
Container-title:American Journal of Respiratory and Critical Care Medicine
language:en
Short-container-title:Am J Respir Crit Care Med

Author:

Bamberg Alison¹²,Redente Elizabeth F.¹³⁴,Groshong Steve D.⁵³,Tuder Rubin M.³,Cool Carlyne D.³,Keith Rebecca C.⁵³,Edelman Benjamin L.¹,Black Bart P.¹,Cosgrove Gregory P.⁵³,Wynes Murry W.¹³,Curran-Everett Douglas⁶,De Langhe Stijn⁷,Ortiz Luis A.⁸,Thorburn Andrew⁹,Riches David W. H.¹²³⁹⁴

Affiliation:

1. Program in Cell Biology, Department of Pediatrics

2. Department of Immunology and Microbiology

3. Division of Pulmonary Sciences and Critical Care Medicine, Department of Medicine, and

4. Department of Research, Veterans Affairs Eastern Colorado Health Care System, Denver, Colorado

5. Department of Medicine, and

6. Division of Biostatistics and Bioinformatics, National Jewish Health, Denver, Colorado

7. Division of Pulmonary, Allergy, and Critical Care Medicine, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama; and

8. Department of Environmental and Occupational Health, University of Pittsburgh, Pittsburgh, Pennsylvania

9. Department of Pharmacology, University of Colorado School of Medicine, Aurora, Colorado

Publisher

American Thoracic Society

Subject

Critical Care and Intensive Care Medicine,Pulmonary and Respiratory Medicine

Link

https://www.atsjournals.org/doi/pdf/10.1164/rccm.201707-1497OC

Reference58 articles.

1. Idiopathic pulmonary fibrosis

2. Prognostic Significance of Histopathologic Subsets in Idiopathic Pulmonary Fibrosis

3. Predicting Survival in Idiopathic Pulmonary Fibrosis

4. Pathogenetic mechanisms in usual interstitial pneumonia/idiopathic pulmonary fibrosis

5. An Essential Role for Resident Fibroblasts in Experimental Lung Fibrosis Is Defined by Lineage-Specific Deletion of High-Affinity Type II Transforming Growth Factor β Receptor

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