Platelet Extracellular Vesicles Drive Inflammasome–IL-1β–Dependent Lung Injury in Sickle Cell Disease-Reference-Cited by-同舟云学术

Platelet Extracellular Vesicles Drive Inflammasome–IL-1β–Dependent Lung Injury in Sickle Cell Disease

Published:2020-01-01 Issue:1 Volume:201 Page:33-46
ISSN:1073-449X
Container-title:American Journal of Respiratory and Critical Care Medicine
language:en
Short-container-title:Am J Respir Crit Care Med

Author:

Vats Ravi¹²,Brzoska Tomasz¹,Bennewitz Margaret F.¹³,Jimenez Maritza A.¹²,Pradhan-Sundd Tirthadipa⁴⁵,Tutuncuoglu Egemen¹,Jonassaint Jude¹⁵,Gutierrez Edgar⁶,Watkins Simon C.⁷,Shiva Sruti¹,Scott Melanie J.⁸,Morelli Adrian E.⁹,Neal Matthew D.⁸,Kato Gregory J.¹⁵,Gladwin Mark T.¹¹⁰,Sundd Prithu¹¹⁰²^ORCID

Affiliation:

1. Pittsburgh Heart, Lung, and Blood Vascular Medicine Institute

2. Department of Bioengineering, University of Pittsburgh, Pittsburgh, Pennsylvania

3. Department of Chemical and Biomedical Engineering, West Virginia University, Morgantown, West Virginia; and

4. Division of Experimental Pathology

5. Division of Hematology and Oncology

6. Department of Physics, University of California San Diego, La Jolla, California

7. Center for Biologic Imaging

8. Department of Surgery

9. Thomas E. Starzl Transplantation Institute, and

10. Division of Pulmonary, Allergy, and Critical Care Medicine, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania

Publisher

American Thoracic Society

Subject

Critical Care and Intensive Care Medicine,Pulmonary and Respiratory Medicine

Link

https://www.atsjournals.org/doi/pdf/10.1164/rccm.201807-1370OC

Reference52 articles.

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2. Global, regional, and national age–sex specific all-cause and cause-specific mortality for 240 causes of death, 1990–2013: a systematic analysis for the Global Burden of Disease Study 2013

3. Sickle cell disease: old discoveries, new concepts, and future promise

4. Pulmonary Complications of Sickle Cell Disease

5. Rapidly progressive acute chest syndrome in individuals with sickle cell anemia: a distinct acute chest syndrome phenotype

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