Caveat: Monoclonal antibodies 20.1 and 103.2 bind all human BTN3A proteins and are not suited to study BTN3A1-specific features
Author:
Affiliation:
1. Institute for Virology and Immunobiology, Julius Maximilians Universität Würzburg, 97078 Würzburg, Germany
Funder
Deutsche Forschungsgemeinschaft
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Link
https://pnas.org/doi/pdf/10.1073/pnas.2304065120
Reference10 articles.
1. Up-regulation of BTN3A1 on CD14+cells promotes Vγ9Vδ2 T cell activation in psoriasis
2. The Molecular Basis for Modulation of Human Vγ9Vδ2 T Cell Responses by CD277/Butyrophilin-3 (BTN3A)-specific Antibodies
3. Butyrophilin 3A/CD277–Dependent Activation of Human γδ T Cells: Accessory Cell Capacity of Distinct Leukocyte Populations
4. Key implication of CD277/butyrophilin-3 (BTN3A) in cellular stress sensing by a major human γδ T-cell subset
5. Heteromeric interactions regulate butyrophilin (BTN) and BTN-like molecules governing γδ T cell biology
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