Loss of chloride channel ClC-5 impairs endocytosis by defective trafficking of megalin and cubilin in kidney proximal tubules-Reference-Cited by-同舟云学术

Loss of chloride channel ClC-5 impairs endocytosis by defective trafficking of megalin and cubilin in kidney proximal tubules

Published:2003-06-18 Issue:14 Volume:100 Page:8472-8477
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:PNAS

Author:

Christensen Erik I.,Devuyst Olivier,Dom Geneviève,Nielsen Rikke,Van Der Smissen Patrick,Verroust Pierre,Leruth Michèle,Guggino William B.,Courtoy Pierre J.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference48 articles.

1. Megalin-dependent cubilin-mediated endocytosis is a major pathway for the apical uptake of transferrin in polarized epithelia

2. The pathogenic antigen of Heymann nephritis is a membrane glycoprotein of the renal proximal tubule brush border.

3. Complete cloning and sequencing of rat gp330/"megalin," a distinctive member of the low density lipoprotein receptor gene family.

4. Identification of rat yolk sac target protein of teratogenic antibodies, gp280, as intrinsic factor-cobalamin receptor.

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