Carboxyl-terminal-truncated apolipoprotein E4 causes Alzheimer's disease-like neurodegeneration and behavioral deficits in transgenic mice-Reference-Cited by-同舟云学术

Carboxyl-terminal-truncated apolipoprotein E4 causes Alzheimer's disease-like neurodegeneration and behavioral deficits in transgenic mice

Published:2003-08-25 Issue:19 Volume:100 Page:10966-10971
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Harris F. M.,Brecht W. J.,Xu Q.,Tesseur I.,Kekonius L.,Wyss-Coray T.,Fish J. D.,Masliah E.,Hopkins P. C.,Scearce-Levie K.,Weisgraber K. H.,Mucke L.,Mahley R. W.,Huang Y.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

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4. Apolipoprotein E: high-avidity binding to beta-amyloid and increased frequency of type 4 allele in late-onset familial Alzheimer disease.

5. Apolipoprotein E Affects the Rate of Alzheimer Disease Expression: β-Amyloid Burden Is a Secondary Consequence Dependent on APOE Genotype and Duration of Disease

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