Disruption of the Jnk2 (Mapk9) gene reduces destructive insulitis and diabetes in a mouse model of type I diabetes-Reference-Cited by-同舟云学术

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Disruption of the Jnk2 (Mapk9) gene reduces destructive insulitis and diabetes in a mouse model of type I diabetes

Published:2005-05-02 Issue:19 Volume:102 Page:6931-6935
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Jaeschke A.,Rincon M.,Doran B.,Reilly J.,Neuberg D.,Greiner D. L.,Shultz L. D.,Rossini A. A.,Flavell R. A.,Davis R. J.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference29 articles.

1. c-Jun N-terminal Kinase (JNK) Mediates Feedback Inhibition of the Insulin Signaling Cascade

2. The c-Jun NH2-terminal Kinase Promotes Insulin Resistance during Association with Insulin Receptor Substrate-1 and Phosphorylation of Ser307

3. An essential role of the JIP1 scaffold protein for JNK activation in adipose tissue

4. A central role for JNK in obesity and insulin resistance

5. The Role of CD4 vs. CD8 T Cells in IDDM

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