Blocking protein farnesyltransferase improves nuclear shape in fibroblasts from humans with progeroid syndromes
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference23 articles.
1. Loss of ZMPSTE24 (FACE-1) causes autosomal recessive restrictive dermopathy and accumulation of Lamin A precursors
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2. The farnesyl transferase inhibitor (FTI) lonafarnib improves nuclear morphology in ZMPSTE24-deficient fibroblasts from patients with the progeroid disorder MAD-B;Nucleus;2023-12-05
3. Use of Farnesyl Transferase Inhibitors in an Ageing Model in Drosophila;Journal of Developmental Biology;2023-10-29
4. Prelamin A and ZMPSTE24 in premature and physiological aging;Nucleus;2023-10-26
5. Progerin, an Aberrant Spliced Form of Lamin A, Is a Potential Therapeutic Target for HGPS;Cells;2023-09-18
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