Endocrine self and gut non-self intersect in the pancreatic lymph nodes
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference28 articles.
1. The Pathogenesis of Insulin-Dependent Diabetes Mellitus
2. Initiation of Autoimmune Diabetes by Developmentally Regulated Presentation of Islet Cell Antigens in the Pancreatic Lymph Nodes
3. Physiological β Cell Death Triggers Priming of Self-reactive T Cells by Dendritic Cells in a Type-1 Diabetes Model
4. Pancreatic Lymph Nodes Are Required for Priming of β Cell Reactive T Cells in NOD Mice
5. Absence of Lymph Nodes in NOD Mice Treated With Lymphotoxin-β Receptor Immunoglobulin Protects From Diabetes
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2. Gut dysbiosis promotes islet-autoimmunity by increasing T-cell attraction in islets via CXCL10 chemokine;Journal of Autoimmunity;2023-11
3. Lymph node sharing between pancreas, gut, and liver leads to immune crosstalk and regulation of pancreatic autoimmunity;Immunity;2023-09
4. Pancreatic draining lymph nodes (PLNs) serve as a pathogenic hub contributing to the development of type 1 diabetes;Cell & Bioscience;2023-08-28
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