The B1-subunit of the H+ ATPase is required for maximal urinary acidification-Reference-Cited by-同舟云学术

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The B1-subunit of the H+ ATPase is required for maximal urinary acidification

Published:2005-09-08 Issue:38 Volume:102 Page:13616-13621
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Finberg K. E.,Wagner C. A.,Bailey M. A.,Paunescu T. G.,Breton S.,Brown D.,Giebisch G.,Geibel J. P.,Lifton R. P.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference20 articles.

1. Mutations in the chloride-bicarbonate exchanger gene AE1 cause autosomal dominant but not autosomal recessive distal renal tubular acidosis

2. Mutations in the gene encoding B1 subunit of H+-ATPase cause renal tubular acidosis with sensorineural deafness

3. Mutations in ATP6N1B, encoding a new kidney vacuolar proton pump 116-kD subunit, cause recessive distal renal tubular acidosis with preserved hearing

4. Physiology and Biochemistry of the Kidney Vacuolar H+-ATPase

5. Renal Vacuolar H+-ATPase

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