Breast cancer bone metastasis mediated by the Smad tumor suppressor pathway-Reference-Cited by-同舟云学术

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Breast cancer bone metastasis mediated by the Smad tumor suppressor pathway

Published:2005-09-19 Issue:39 Volume:102 Page:13909-13914
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Kang Y.,He W.,Tulley S.,Gupta G. P.,Serganova I.,Chen C.-R.,Manova-Todorova K.,Blasberg R.,Gerald W. L.,Massague J.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference36 articles.

1. TGF-β signaling in tumor suppression and cancer progression

2. Cytostatic and apoptotic actions of TGF-β in homeostasis and cancer

3. Mechanisms of TGF-β Signaling from Cell Membrane to the Nucleus

4. Molecular Predictors of Survival after Adjuvant Chemotherapy for Colon Cancer

5. Transforming growth factor β signaling impairs Neu-induced mammary tumorigenesis while promoting pulmonary metastasis

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1. C1orf106 (INAVA) Is a SMAD3-Dependent TGF-β Target Gene That Promotes Clonogenicity and Correlates with Poor Prognosis in Breast Cancer;Cells;2024-09-12

2. Sarcomatoid carcinoma of the pancreas (Review);Oncology Letters;2024-08-05

3. Verteporfin inhibits TGF-β signaling by disrupting the Smad2/3–Smad4 interaction;Molecular Biology of the Cell;2024-07-01

4. Comprehensive molecular interaction map of TGFβ induced epithelial to mesenchymal transition in breast cancer;npj Systems Biology and Applications;2024-05-17

5. From Tumor to Bone: Growth Factor Receptors as Key Players in Cancer Metastasis;Frontiers in Bioscience-Landmark;2024-05-13

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