Nonimmunoglobulin target loci of activation-induced cytidine deaminase (AID) share unique features with immunoglobulin genes-Reference-Cited by-同舟云学术

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Nonimmunoglobulin target loci of activation-induced cytidine deaminase (AID) share unique features with immunoglobulin genes

Published:2012-01-30 Issue:7 Volume:109 Page:2479-2484
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Kato L.,Begum N. A.,Burroughs A. M.,Doi T.,Kawai J.,Daub C. O.,Kawaguchi T.,Matsuda F.,Hayashizaki Y.,Honjo T.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference61 articles.

1. Class Switch Recombination and Hypermutation Require Activation-Induced Cytidine Deaminase (AID), a Potential RNA Editing Enzyme

2. Activation-Induced Cytidine Deaminase (AID) Deficiency Causes the Autosomal Recessive Form of the Hyper-IgM Syndrome (HIGM2)

3. Molecular Mechanism of Class Switch Recombination: Linkage with Somatic Hypermutation

4. Immunoglobulin Somatic Hypermutation

5. Separate domains of AID are required for somatic hypermutation and class-switch recombination

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