Glut9 is a major regulator of urate homeostasis and its genetic inactivation induces hyperuricosuria and urate nephropathy-Reference-Cited by-同舟云学术

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Glut9 is a major regulator of urate homeostasis and its genetic inactivation induces hyperuricosuria and urate nephropathy

Published:2009-08-21 Issue:36 Volume:106 Page:15501-15506
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Preitner F.,Bonny O.,Laverriere A.,Rotman S.,Firsov D.,Da Costa A.,Metref S.,Thorens B.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference28 articles.

1. Uric Acid and Cardiovascular Risk

2. Uric acid: bystander or culprit in hypertension and progressive renal disease?

3. The inflammasome: a danger sensing complex triggering innate immunity

4. Gout-associated uric acid crystals activate the NALP3 inflammasome

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1. Regulation of Urate Homeostasis by Membrane Transporters;Gout, Urate, and Crystal Deposition Disease;2024-06-19

2. Structural basis for urate recognition and apigenin inhibition of human GLUT9;Nature Communications;2024-06-12

3. CRISPR/Cas9 Mediated Deletion of the Uox Gene Generates a Mouse Model of Hyperuricemia with Multiple Complications;Journal of Cardiovascular Translational Research;2024-06-10

4. Role of Transporters in Hyperuricemia;Physiology;2024-05-22

5. Signaling pathways in uric acid homeostasis and gout: From pathogenesis to therapeutic interventions;International Immunopharmacology;2024-05

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