CD39 deletion exacerbates experimental murine colitis and human polymorphisms increase susceptibility to inflammatory bowel disease-Reference-Cited by-同舟云学术

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CD39 deletion exacerbates experimental murine colitis and human polymorphisms increase susceptibility to inflammatory bowel disease

Published:2009-09-28 Issue:39 Volume:106 Page:16788-16793
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Friedman D. J.,Kunzli B. M.,A-Rahim Y. I.,Sevigny J.,Berberat P. O.,Enjyoji K.,Csizmadia E.,Friess H.,Robson S. C.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference40 articles.

1. The genetics and immunopathogenesis of inflammatory bowel disease

2. Ulcerative colitis and Crohn's disease in an unselected population of monozygotic and dizygotic twins. A study of heritability and the influence of smoking.

3. Adenosine generation catalyzed by CD39 and CD73 expressed on regulatory T cells mediates immune suppression

4. CD39/NTPDase-1 activity and expression in normal leukocytes

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