Ablation of triadin causes loss of cardiac Ca2+ release units, impaired excitation-contraction coupling, and cardiac arrhythmias-Reference-Cited by-同舟云学术

Ablation of triadin causes loss of cardiac Ca2+ release units, impaired excitation-contraction coupling, and cardiac arrhythmias

Published:2009-04-21 Issue:18 Volume:106 Page:7636-7641
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Chopra N.,Yang T.,Asghari P.,Moore E. D.,Huke S.,Akin B.,Cattolica R. A.,Perez C. F.,Hlaing T.,Knollmann-Ritschel B. E. C.,Jones L. R.,Pessah I. N.,Allen P. D.,Franzini-Armstrong C.,Knollmann B. C.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference34 articles.

1. Formation of junctions involved in excitation-contraction coupling in skeletal and cardiac muscle.

2. Macromolecular complexes regulating cardiac ryanodine receptor function

3. Complex Formation between Junctin, Triadin, Calsequestrin, and the Ryanodine Receptor

4. Biochemical Characterization and Molecular Cloning of Cardiac Triadin

5. Localization and partial characterization of the oligomeric disulfide-linked molecular weight 95,000 protein (triadin) which binds the ryanodine and dihydropyridine receptors in skeletal muscle triadic vesicles

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