beta Cells cannot directly prime diabetogenic CD8 T cells in nonobese diabetic mice
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference22 articles.
1. Transgenic Expression of Dominant-Negative Fas-Associated Death Domain Protein in β Cells Protects against Fas Ligand-Induced Apoptosis and Reduces Spontaneous Diabetes in Nonobese Diabetic Mice
2. Reduced Incidence and Delayed Onset of Diabetes in Perforin-deficient Nonobese Diabetic Mice
3. Major histocompatibility complex class I molecules are required for the development of insulitis in non-obese diabetic mice
4. Adoptive Transfer of Diabetes Into Immunodeficient NOD-scid/scid Mice: Relative Contributions of CD4+ and CD8+ T-Cells From Diabetic Versus Prediabetic NOD.NON-Thy-1a Donors
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1. The Application of High‐Throughput Approaches in Identifying Novel Therapeutic Targets and Agents to Treat Diabetes;Advanced Biology;2022-11-18
2. Therapeutic Strategies for Diabetes: Immune Modulation in Pancreatic β Cells;Frontiers in Endocrinology;2021-08-17
3. Not-so-opposite ends of the spectrum: CD8+ T cell dysfunction across chronic infection, cancer and autoimmunity;Nature Immunology;2021-06-17
4. Dietary SCFAs, IL-22, and GFAP: The Three Musketeers in the Gut–Neuro–Immune Network in Type 1 Diabetes;Frontiers in Immunology;2019-10-29
5. Pancreatic ductal cell antigens are important in the development of invasive insulitis in Non-Obese Diabetic mice;Journal of Neuroimmunology;2019-02
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