A low-molecular-weight compound discovered through virtual database screening inhibits Stat3 function in breast cancer cells
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference33 articles.
1. Jak-STAT Pathways and Transcriptional Activation in Response to IFNs and Other Extracellular Signaling Proteins
2. Stat3 Activation Is Required for Cellular Transformation by v- src
3. Constitutive activation of Stat3 by the Src and JAK tyrosine kinases participates in growth regulation of human breast carcinoma cells
4. Stat3: a STAT Family Member Activated by Tyrosine Phosphorylation in Response to Epidermal Growth Factor and Interleukin-6
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