Postmenopausal osteoporosis, T cells, and immune dysfunction
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference19 articles.
1. Origin of osteoclasts: mature monocytes and macrophages are capable of differentiating into osteoclasts under a suitable microenvironment prepared by bone marrow-derived stromal cells.
2. Osteoprotegerin Ligand Is a Cytokine that Regulates Osteoclast Differentiation and Activation
3. Spontaneous release of interleukin 1 from human blood monocytes reflects bone formation in idiopathic osteoporosis.
4. Human blood-mobilized hematopoietic precursors differentiate into osteoclasts in the absence of stromal cells.
5. Estrogen deficiency induces bone loss by enhancing T-cell production of TNF-α
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