Maintenance of adenomatous polyposis coli (APC)-mutant colorectal cancer is dependent on Wnt/ -catenin signaling-Reference-Cited by-同舟云学术

Maintenance of adenomatous polyposis coli (APC)-mutant colorectal cancer is dependent on Wnt/ -catenin signaling

Published:2011-09-23 Issue:41 Volume:108 Page:17135-17140
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Scholer-Dahirel A.,Schlabach M. R.,Loo A.,Bagdasarian L.,Meyer R.,Guo R.,Woolfenden S.,Yu K. K.,Markovits J.,Killary K.,Sonkin D.,Yao Y.-M.,Warmuth M.,Sellers W. R.,Schlegel R.,Stegmeier F.,Mosher R. E.,McLaughlin M. E.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference30 articles.

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2. Constitutive Transcriptional Activation by a β-Catenin-Tcf Complex in APC −/− Colon Carcinoma

3. Activation of β-Catenin-Tcf Signaling in Colon Cancer by Mutations in β-Catenin or APC

4. Stabilization of β-Catenin by Genetic Defects in Melanoma Cell Lines

5. Mutations in AXIN2 cause colorectal cancer with defective mismatch repair by activating β-catenin/TCF signalling

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