The V122I cardiomyopathy variant of transthyretin increases the velocity of rate-limiting tetramer dissociation, resulting in accelerated amyloidosis
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference45 articles.
1. Alternative conformations of amyloidogenic proteins govern their behavior
2. The Genetics of the Amyloidoses
3. Protein misfolding, evolution and disease
4. Protein aggregation: folding aggregates, inclusion bodies and amyloid
5. The pathogenesis and biochemistry of amyloidosis
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