Interleukin (IL) 1 , IL-1 receptor antagonist, IL-10, and IL-13 gene expression in the central nervous system and anterior pituitary during systemic inflammation: Pathophysiological implications-Reference-Cited by-同舟云学术

Interleukin (IL) 1 , IL-1 receptor antagonist, IL-10, and IL-13 gene expression in the central nervous system and anterior pituitary during systemic inflammation: Pathophysiological implications

Published:1997-01-07 Issue:1 Volume:94 Page:227-232
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Wong M.-L.,Bongiorno P. B.,Rettori V.,McCann S. M.,Licinio J.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference48 articles.

1. Cytokines and the nervous system II: actions and mechanisms of action

2. Resistance to fever induction and impaired acute-phase response in interleukin-1β-deficient mice

3. A mechanism for the inhibition of fever by a virus.

4. Balance of synovial fluid IL-1β and IL-1 receptor antagonist and recovery from Lyme arthritis

5. Interleukin-1β specifically stimulates nitric oxide production in the hypothalamus

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