DDB2 gene disruption leads to skin tumors and resistance to apoptosis after exposure to ultraviolet light but not a chemical carcinogen
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference38 articles.
1. Reinvestigation of the Classification of Five Cell Strains of Xeroderma Pigmentosum Group E with Reclassification of Three of Them
2. Abnormal regulation of DDB2 gene expression in xeroderma pigmentosum group E strains
3. True XP group E patients have a defective UV-damaged DNA binding protein complex and mutations in DDB2 which reveal the functional domains of its p48 product
4. Characterization of a human DNA damage binding protein implicated in xeroderma pigmentosum E.
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4. USP44 Stabilizes DDB2 to Facilitate Nucleotide Excision Repair and Prevent Tumors;Frontiers in Cell and Developmental Biology;2021-04-16
5. Expanding molecular roles of UV-DDB: Shining light on genome stability and cancer;DNA Repair;2020-10
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