Dysregulation of gene expression as a cause of Cockayne syndrome neurological disease-Reference-Cited by-同舟云学术

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Dysregulation of gene expression as a cause of Cockayne syndrome neurological disease

Published:2014-09-23 Issue:40 Volume:111 Page:14454-14459
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proc Natl Acad Sci USA

Author:

Wang Yuming,Chakravarty Probir,Ranes Michael,Kelly Gavin,Brooks Philip J.,Neilan Edward,Stewart Aengus,Schiavo Giampietro,Svejstrup Jesper Q.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference37 articles.

1. Neuropathology of Cockayne syndrome: Evidence for impaired development, premature aging, and neurodegeneration

2. Blinded by the UV light: How the focus on transcription-coupled NER has distracted from understanding the mechanisms of Cockayne syndrome neurologic disease

3. Mechanisms of transcription-coupled DNA repair

4. Transcription-coupled DNA repair: two decades of progress and surprises

5. Mammalian Transcription-Coupled Excision Repair

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1. General loss of proteostasis links Huntington disease to Cockayne syndrome;Neurobiology of Disease;2024-09

2. Hao-Fountain syndrome protein USP7 controls neuronal differentiation via BCOR-ncPRC1.1;2024-08-04

3. Cognitive Decline and Other Late-Stage Neurologic Complications in Cockayne Syndrome;Neurology Clinical Practice;2024-08

4. Differential processing of RNA polymerase II at DNA damage correlates with transcription-coupled repair syndrome severity;Nucleic Acids Research;2024-07-18

5. Cockayne Syndrome Linked to Elevated R-Loops Induced by Stalled RNA Polymerase II during Transcription Elongation;Nature Communications;2024-07-17

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