Inhibition of the leucine-rich repeat protein LINGO-1 enhances survival, structure, and function of dopaminergic neurons in Parkinson's disease models-Reference-Cited by-同舟云学术

Inhibition of the leucine-rich repeat protein LINGO-1 enhances survival, structure, and function of dopaminergic neurons in Parkinson's disease models

Published:2007-08-28 Issue:36 Volume:104 Page:14430-14435
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Inoue H.,Lin L.,Lee X.,Shao Z.,Mendes S.,Snodgrass-Belt P.,Sweigard H.,Engber T.,Pepinsky B.,Yang L.,Beal M. F.,Mi S.,Isacson O.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference35 articles.

1. Transcription-dependent and -independent control of neuronal survival by the PI3K–Akt signaling pathway

2. Influences of dopaminergic lesion on epidermal growth factor-ErbB signals in Parkinson's disease and its model: neurotrophic implication in nigrostriatal neurons

3. Ex vivogene delivery of GDNF using primary astrocytes transduced with a lentiviral vector provides neuroprotection in a rat model of Parkinson's disease

4. Heparin-binding epidermal growth factor-like growth factor: a component in chromaffin granules which promotes the survival of nigrostriatal dopaminergic neurones in vitro and in vivo

5. Brain-derived growth factor and glial cell line-derived growth factor use distinct intracellular signaling pathways to protect PD cybrids from HO-induced neuronal death

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