Loss of the Par-1b/MARK2 polarity kinase leads to increased metabolic rate, decreased adiposity, and insulin hypersensitivity in vivo-Reference-Cited by-同舟云学术

Loss of the Par-1b/MARK2 polarity kinase leads to increased metabolic rate, decreased adiposity, and insulin hypersensitivity in vivo

Published:2007-03-19 Issue:13 Volume:104 Page:5680-5685
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Hurov J. B.,Huang M.,White L. S.,Lennerz J.,Choi C. S.,Cho Y.-R.,Kim H.-J.,Prior J. L.,Piwnica-Worms D.,Cantley L. C.,Kim J. K.,Shulman G. I.,Piwnica-Worms H.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference56 articles.

1. par-1, a gene required for establishing polarity in C. elegans embryos, encodes a putative Ser/Thr kinase that is asymmetrically distributed

2. The Yeast Par-1 Homologs Kin1 and Kin2 Show Genetic and Physical Interactions with Components of the Exocytic Machinery

3. The protein kinase kin1, the fission yeast orthologue of mammalian MARK/PAR-1, localises to new cell ends after mitosis and is important for bipolar growth

4. Drosophila par-1 is required for oocyte differentiation and microtubule organization

5. Bazooka and PAR-6 are required with PAR-1 for the maintenance of oocyte fate in Drosophila

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