Defects in XRCC4 and KU80 differentially affect the joining of distal nonhomologous ends
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference43 articles.
1. Impact of the KU80 Pathway on NHEJ-Induced Genome Rearrangements in Mammalian Cells
2. Repair of DNA double strand breaks by non-homologous end joining
3. Cernunnos-XLF, a recently identified non-homologous end-joining factor required for the development of the immune system
4. DNA double-strand break repair in cell-free extracts from Ku80-deficient cells: implications for Ku serving as an alignment factor in non-homologous DNA end joining
5. Double-strand break repair in Ku86- and XRCC4-deficient cells
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2. Uncovering the dynamics of precise repair at CRISPR/Cas9-induced double-strand breaks;Nature Communications;2024-06-14
3. The cGAS-Ku80 complex regulates the balance between two end joining subpathways;Cell Death & Differentiation;2024-04-25
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5. Proximal binding of dCas9 at a DNA double strand break stimulates homology-directed repair as a local inhibitor of classical non-homologous end joining;Nucleic Acids Research;2023-03-02
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