Molecular switch for CLC-K Cl- channel block/activation: Optimal pharmacophoric requirements towards high-affinity ligands-Reference-Cited by-同舟云学术

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Molecular switch for CLC-K Cl- channel block/activation: Optimal pharmacophoric requirements towards high-affinity ligands

Published:2008-01-23 Issue:4 Volume:105 Page:1369-1373
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Liantonio A.,Picollo A.,Carbonara G.,Fracchiolla G.,Tortorella P.,Loiodice F.,Laghezza A.,Babini E.,Zifarelli G.,Pusch M.,Camerino D. C.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference26 articles.

1. Chloride Transport in the Kidney: Lessons from Human Disease and Knockout Mice

2. FUNCTION OF CHLORIDE CHANNELS IN THE KIDNEY

3. Mutations in the chloride channel gene, CLCNKB, cause Bartter's syndrome type III

4. Barttin is a Cl- channel β-subunit crucial for renal Cl- reabsorption and inner ear K+ secretion

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3. ClC-K Kidney Chloride Channels: From Structure to Pathology;Anion Channels and Transporters;2023

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5. THE CONCISE GUIDE TO PHARMACOLOGY 2021/22: Ion channels;British Journal of Pharmacology;2021-09-16

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