IL-18 contributes to the spontaneous development of atopic dermatitis-like inflammatory skin lesion independently of IgE/stat6 under specific pathogen-free conditions-Reference-Cited by-同舟云学术

IL-18 contributes to the spontaneous development of atopic dermatitis-like inflammatory skin lesion independently of IgE/stat6 under specific pathogen-free conditions

Published:2002-07-31 Issue:17 Volume:99 Page:11340-11345
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Konishi H.,Tsutsui H.,Murakami T.,Yumikura-Futatsugi S.,Yamanaka K.-i.,Tanaka M.,Iwakura Y.,Suzuki N.,Takeda K.,Akira S.,Nakanishi K.,Mizutani H.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference44 articles.

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2. Atopic dermatitis

3. Mediators of Immediate Hypersensitivity Reactions

4. Mast cell lines produce lymphokines in response to cross-linkage of FcεRI or to calcium ionophores

5. Lymphokine and Cytokine Production by FcεRI+ Cells

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