-Amyloid peptides enhance -synuclein accumulation and neuronal deficits in a transgenic mouse model linking Alzheimer's disease and Parkinson's disease-Reference-Cited by-同舟云学术

-Amyloid peptides enhance -synuclein accumulation and neuronal deficits in a transgenic mouse model linking Alzheimer's disease and Parkinson's disease

Published:2001-09-25 Issue:21 Volume:98 Page:12245-12250
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Masliah E.,Rockenstein E.,Veinbergs I.,Sagara Y.,Mallory M.,Hashimoto M.,Mucke L.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

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1. Neuropathologic and clinical features of Parkinson's disease in Alzheimer's disease patients

2. The Lewy body variant of Alzheimer's disease: A clinical and pathologic entity

3. Clinical-Neuropathological Correlations in Alzheimer's Disease and Related Dementias

4. Parkinson's Disease

5. Mechanisms contributing to the deficits in hippocampal synaptic plasticity in mice lacking amyloid precursor protein

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