Deletion of claudin-10 (Cldn10) in the thick ascending limb impairs paracellular sodium permeability and leads to hypermagnesemia and nephrocalcinosis-Reference-Cited by-同舟云学术

Deletion of claudin-10 (Cldn10) in the thick ascending limb impairs paracellular sodium permeability and leads to hypermagnesemia and nephrocalcinosis

Published:2012-08-13 Issue:35 Volume:109 Page:14241-14246
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Breiderhoff T.,Himmerkus N.,Stuiver M.,Mutig K.,Will C.,Meij I. C.,Bachmann S.,Bleich M.,Willnow T. E.,Muller D.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference26 articles.

1. Bartter- and Gitelman-like syndromes: salt-losing tubulopathies with loop or DCT defects

2. Cation selectivity of the isolated perfused cortical thick ascending limb of Henle's loop of rabbit kidney

3. Molecular Basis of the Core Structure of Tight Junctions

4. Predicted expansion of the claudin multigene family

5. Claudins create charge-selective channels in the paracellular pathway between epithelial cells

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