Quantitative modeling of the terminal differentiation of B cells and mechanisms of lymphomagenesis-Reference-Cited by-同舟云学术

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Quantitative modeling of the terminal differentiation of B cells and mechanisms of lymphomagenesis

Published:2012-01-30 Issue:7 Volume:109 Page:2672-2677
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Martinez M. R.,Corradin A.,Klein U.,Alvarez M. J.,Toffolo G. M.,di Camillo B.,Califano A.,Stolovitzky G. A.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference42 articles.

1. Mechanism of antigen-driven selection in germinal centres

2. Control of Inflammation, Cytokine Expression, and Germinal Center Formation by BCL-6

3. Antigen receptor signaling induces MAP kinase-mediated phosphorylation and degradation of the BCL-6 transcription factor

4. Requirement for the Transcription Factor LSIRF/IRF4 for Mature B and T Lymphocyte Function

5. A Signaling Pathway Mediating Downregulation of BCL6 in Germinal Center B Cells Is Blocked by BCL6 Gene Alterations in B Cell Lymphoma

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1. A multiscale spatial modeling framework for the germinal center response;Frontiers in Immunology;2024-05-30

2. Stochastic modeling of a gene regulatory network driving B cell development in germinal centers;PLOS ONE;2024-03-28

3. A Multiscale Spatial Modeling Framework for the Germinal Center Response;2024-01-30

4. Frequent ZNF217 mutations lead to transcriptional deregulation of interferon signal transduction via altered chromatin accessibility in B cell lymphoma;Leukemia;2023-08-30

5. New tools for immunologists: models of lymph node function from cells to tissues;Frontiers in Immunology;2023-05-10

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