Signal transducer and activator of transcription 3 (STAT3) and survivin induction by varicella-zoster virus promote replication and skin pathogenesis
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference40 articles.
1. Varicella-Zoster Virus T Cell Tropism and the Pathogenesis of Skin Infection
2. Varicella-zoster virus infection of human dorsal root ganglia in vivo
3. Varicella-Zoster Virus Immediate-Early Protein 62 Blocks Interferon Regulatory Factor 3 (IRF3) Phosphorylation at Key Serine Residues: a Novel Mechanism of IRF3 Inhibition among Herpesviruses
4. Inhibition of the NF-κB Pathway by Varicella-Zoster Virus In Vitro and in Human Epidermal Cells In Vivo
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1. STAT3 Increases CVB3 Replication and Acute Pancreatitis and Myocarditis Pathology via Impeding Nuclear Translocation of STAT1 and Interferon-Stimulated Gene Expression;International Journal of Molecular Sciences;2024-08-19
2. IL-6/STAT3 axis is hijacked by GCRV to facilitate viral replication via suppressing type Ⅰ IFN signaling;Fish & Shellfish Immunology;2024-06
3. Direct and biologically significant interactions of human herpesvirus 8 interferon regulatory factor 1 with STAT3 and Janus kinase TYK2;PLOS Pathogens;2023-11-20
4. Double-edged sword of JAK/STAT signaling pathway in viral infections: novel insights into virotherapy;Cell Communication and Signaling;2023-10-02
5. Creating the “Dew Drop on a Rose Petal”: the Molecular Pathogenesis of Varicella-Zoster Virus Skin Lesions;Microbiology and Molecular Biology Reviews;2023-09-26
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